01 · PUMP & FILLING
Output and filling are linked.
Heart failure is a clinical syndrome in which the heart cannot meet circulatory demands without raised filling pressures or compensatory change. Start with pump performance, filling and circulation.
Retrieve: How can impaired output and raised filling pressure coexist?
02 · CONGESTION
Where does fluid pressure show?
Congestion questions connect upstream pressure with the vascular bed affected. Symptoms and signs are clues to haemodynamics, not isolated checkboxes.
Retrieve: What is the physiological bridge between raised filling pressure and congestion?
03 · PERFUSION
Low flow has a pattern.
Reduced forward output affects perfusion and may provoke compensatory responses. Distinguish evidence of congestion from evidence of reduced delivery; both can be present.
Retrieve: Which clues would make you think about reduced forward flow?
04 · COMPENSATION
Adaptation can become burden.
Neurohormonal and structural responses may initially support circulation but can contribute to remodelling and progression. The high-yield idea is dynamic compensation.
Retrieve: Why can a compensatory response become maladaptive over time?
05 · PHENOTYPE
Function is a spectrum.
Questions may organise phenotype through ventricular function, structure, cause, rhythm, valves or time course. One measure is useful evidence, but it is not the whole syndrome.
Retrieve: Why is a single function measure not a complete patient story?
06 · EVIDENCE
Every test has a job.
History, examination, ECG, imaging, biomarkers and laboratory context contribute different pieces. Ask what each finding supports, challenges or leaves unanswered.
Retrieve: Name four evidence domains that contribute to a heart-failure vignette.